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JAC Advance Access originally published online on February 27, 2009
Journal of Antimicrobial Chemotherapy 2009 63(5):1071-1073; doi:10.1093/jac/dkp052
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© The Author 2009. Published by Oxford University Press on behalf of the British Society for Antimicrobial Chemotherapy. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

Research letters

Diversity of plasmid-mediated carbapenem-hydrolysing oxacillinases among carbapenem-resistant Acinetobacter baumannii isolates from Kingdom of Bahrain

Pauline D. Mugnier1, Khalid Mubarak Bindayna2, Laurent Poirel1 and Patrice Nordmann1,*

1 Service de Bactériologie-Virologie, INSERM U914, Emerging Resistance to Antibiotics, Hôpital de Bicêtre, Assistance Publique/Hôpitaux de Paris, Faculté de Médecine et Université Paris Sud, Bicêtre, France 2 College of Medecine, Arabian Gulf University and Salmaniya Medical Complex, Kingdom of Bahrain

* Corresponding author. Service de Bactériologie-Virologie, Hôpital de Bicêtre, 76 rue du général Leclerc, 94275 Le Kremlin-Bicêtre cedex, France. Tel: +33-1-45-21-36-24; Fax: +33-1-45-21-63-40; E-mail: nordmann.patrice@bct.aphp.fr

Keywords: A. baumannii , Middle East , resistance , β-lactamases

The first 10% of the full text of this article appears below.

Sir,

Carbapenem resistance in Acinetobacter baumannii is now increasingly reported. The most prevalent mechanism of carbapenem resistance in A. baumannii corresponds to the production of acquired carbapenem-hydrolysing class D β-lactamases (CHDLs)1 encoded by blaOXA-23-like, blaOXA-40-like or blaOXA-58-like genes. Whereas the blaOXA-40 gene has been detected in Portugal, Spain and the USA, blaOXA-23 and blaOXA-58 genes have disseminated worldwide.1

The aim of our study was to identify the molecular mechanisms leading to carbapenem resistance among a panel of A. baumannii isolates exhibiting resistance or intermediate susceptibility to imipenem and collected from September 2007 to March 2008 . . . [Full Text of this Article]


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