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JAC Advance Access originally published online on July 31, 2008
Journal of Antimicrobial Chemotherapy 2008 62(5):1151-1152; doi:10.1093/jac/dkn295
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© The Author 2008. Published by Oxford University Press on behalf of the British Society for Antimicrobial Chemotherapy. All rights reserved. For Permissions, please e-mail: journals.permissions@oxfordjournals.org

Research letters

First detection of plasmid-mediated quinolone resistance in the community setting and in hospitalized patients in Switzerland

Nadia Liassine1,{dagger}, Patricia Zulueta-Rodriguez2,{ddagger}, Céline Corbel2, Christine Lascols2, Claude-James Soussy2,3 and Emmanuelle Cambau2,3,*

1 Unilabs Genève, Laboratoire de Microbiologie, Genève, Switzerland 2 Université Paris 12, IFR10, Créteil F-94010, France 3 AP-HP, Groupe Hospitalier Henri Mondor-Albert Chenevier, Bactériologie-Virologie-Hygiène, Créteil F-94010, France

* Correspondence address: Bacteriologie, CHU Henri Mondor, 51 Avenue du Marechal de Lattre de Tassigny, 94010 Créteil Cedex, France. Tel: +33-149812831; Fax: +33-149812839; E-mail: emmanuelle.cambau@hmn.aphp.fr

Keywords: fluoroquinolones , qnr , multidrug resistance screening

The first 10% of the full text of this article appears below.

Sir,

Plasmid-mediated quinolone resistance described so far is mainly due to qnr genes (qnrA, qnrB, qnrS and their variants).1 qnr-positive (qnr+) strains were isolated as early as 1994, but mainly from 2001 onwards in strains producing extended- spectrum β-lactamases (ESBLs).1 No strain has been reported to date in Switzerland, although ESBL-producing Enterobacteriaceae are prevalent and qnr+ isolates were reported in France and Germany, which border Switzerland.1,2 In contrast to previous prevalence studies that have been conducted in the hospital setting, we screened isolates . . . [Full Text of this Article]


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