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JAC Advance Access originally published online on December 4, 2003
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Journal of Antimicrobial Chemotherapy (2004) 53, 118-119
© 2004 The British Society for Antimicrobial Chemotherapy

Correspondence

Detection of tet(M), tet(O) and tet(S) in tetracycline/minocycline-resistant Streptococcus pyogenes bacteraemia isolates

Anette M. Hammerum1,*, Hans Ulrik K. Nielsen1, Yvonne Agersø2, Kim Ekelund3 and Niels Frimodt-Moller1

1 National Center for Antimicrobials and Infection Control, Statens Serum Institut, 5 Artillerivej, DK-2300 Copenhagen S; 2 Danish Veterinary Institute, 27 Bülowsvej, DK-1790 Copenhagen V; 3 Streptococcus Unit, Department of Respiratory Infections, Meningitis and STIs, Statens Serum Institut, 5 Artillerivej, DK-2300 Copenhagen S, Denmark

Keywords: PFGE, T typing, gene transfer

The first 10% of the full text of this article appears below.

Sir,

Streptococcus pyogenes is the most common cause of bacterial pharyngitis and causes scarlet fever, impetigo and erysipelas. S. pyogenes may also give rise to severe invasive manifesttions such as sepsis, necrotizing fasciitis and streptococcal toxic shock syndrome.

Tetracycline/minocycline resistance is often encoded by the tet(M) gene in Gram-positive bacteria, and more rarely by the tet(O), tet(Q), tet(S), tet(T) and tet(W) genes, which all encode ribosomal protection proteins.1 Tetracycline resistance (TcR) alone is often encoded by the efflux genes tet(K) and . . . [Full Text of this Article]

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