Skip Navigation

This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF) Freely available
Right arrow Alert me when this article is cited
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (10)
Right arrowRequest Permissions
Right arrow Disclaimer
Google Scholar
Right arrow Articles by Portillo, A.
Right arrow Articles by Torres, C.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Portillo, A.
Right arrow Articles by Torres, C.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

Journal of Antimicrobial Chemotherapy (2001) 47, 115-116
© 2001 The British Society for Antimicrobial Chemotherapy


Correspondence

MLS resistance phenotypes and mechanisms in ß-haemolytic group B, C and G Streptococcusisolates in La Rioja, Spain

A. Portilloa, M. Lanterob, I. Olarteb, F. Ruiz-Larreaa and C. Torresa,*

a Area Bioquímica y Biología Molecular, Universidad de La Rioja, Madre de Dios 51, 26006 Logroño, Spain; b Laboratorio Microbiología, Hospital San Millán, Logroño, Spain

Sir,

Two main mechanisms of resistance to macrolidelincosamide-streptogramin (MLS) antibiotics have been described in group B (GBS), group C (GCS) and group G (GGS) ß-haemolytic streptococci. Target site modification by rRNA methylases, encoded by erm genes, confers the MLSB resistance phenotype [erythromycin-resistant– clindamycin resistant (ERYR-CLIR)]. The mechanism of active efflux confers a dissociate M resistance phenotype [erythromycin-resistant–clindamycin-susceptible (ERYR-CLIS)]. The objective of the present study was to determine the incidence of MLS resistance in vitro among 136 GBS, eight GCS and 28 GGS consecutive clinical isolates recovered from the Hospital San Millán . . . [Full Text of this Article]

Acknowledgments

Notes

References


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
J. Clin. Microbiol.Home page
H. A. Lopardo, P. Vidal, P. Jeric, D. Centron, H. Paganini, R. R. Facklam, and J. Elliott
Six-Month Multicenter Study on Invasive Infections Due to Group B Streptococci in Argentina
J. Clin. Microbiol., October 1, 2003; 41(10): 4688 - 4694.
[Abstract] [Full Text] [PDF]


Home page
J Antimicrob ChemotherHome page
H. Seppala, M. Haanpera, M. Al-Juhaish, H. Jarvinen, J. Jalava, and P. Huovinen
Antimicrobial susceptibility patterns and macrolide resistance genes of viridans group streptococci from normal flora
J. Antimicrob. Chemother., October 1, 2003; 52(4): 636 - 644.
[Abstract] [Full Text] [PDF]


Home page
J Antimicrob ChemotherHome page
A. Portillo, M. J. Gastanares, F. Ruiz-Larrea, and C. Torres
Clonal diversity among erythromycin-resistant {beta}-haemolytic Streptococcus isolates in La Rioja, Spain
J. Antimicrob. Chemother., September 1, 2003; 52(3): 485 - 488.
[Abstract] [Full Text] [PDF]


Home page
Antimicrob. Agents Chemother.Home page
E. Culebras, I. Rodriguez-Avial, C. Betriu, M. Redondo, and J. J. Picazo
Macrolide and Tetracycline Resistance and Molecular Relationships of Clinical Strains of Streptococcus agalactiae
Antimicrob. Agents Chemother., May 1, 2002; 46(5): 1574 - 1576.
[Abstract] [Full Text] [PDF]


Home page
Antimicrob. Agents Chemother.Home page
D. De Mouy, J.-D. Cavallo, R. Leclercq, R. Fabre, and The Aforcopi-Bio Network
Antibiotic Susceptibility and Mechanisms of Erythromycin Resistance in Clinical Isolates of Streptococcus agalactiae: French Multicenter Study
Antimicrob. Agents Chemother., August 1, 2001; 45(8): 2400 - 2402.
[Abstract] [Full Text] [PDF]