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Journal of Antimicrobial Chemotherapy (2000) 46, 145-146
© 2000 The British Society for Antimicrobial Chemotherapy

Correspondence

Prevalence of two different genes encoding NorA in 23 clinical strains of Staphylococcus aureus

Josep M. Sierra, Joaquim Ruiz, M. T. Jimenez De Anta and Jordi Vila*

Department of Microbiology, IDIBAPS, Hospital Clinic, School of Medicine, University of Barcelona, Villarroel 170, 08036 Barcelona, Spain

Sir,

The mechanism of resistance to quinolones in Staphylococcus aureus results from the presence of several mutations in genes encoding DNA gyrase and topoisomerase IV or from overexpression of efflux pumps, such as NorA.1,2 The most important point mutations associated with the acquisition of quinolone resistance are in the gyrA and grlA genes,1 which encode the A subunit of DNA gyrase and topoisomerase IV, respectively. NorA plays an important role in the . . . [Full Text of this Article]

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